Ⅵ 結論
高周波電磁波ばく露が血清エストロゲンに及ぼす影響、及びエストロゲン様作用を有するかをラットの血清エストラジオール、子宮重量を測定することにより検討する実験において、脳平均SAR7.5W/kgの1439MHz TDMA PDC方式の電磁波を1日4時間、3日間連続でばく露させたが、有意差は認められなかった。
携帯電話から発生される高周波電磁波のレベル（局所平均SAR＜2.0W/kg）を大幅に上回るばく露によっても、卵巣切除後ラットにおいて血清エストラジオール、子宮重量には有意差が認められなかった。
Ⅶ 文献
1. Clemons M, Goss P: Estrogen and the risk of breast cancer. N Engl J Med2001;344:276-285.
2. Miller M: DES research heats up again after breast cancer finding. J Natl
Cancer Inst 1999;91:1361-1363.
3. Erren TC: A meta-analysis of epidemiologic studies of electric and magneticfields and breast cancer in women and men. Bioelectromagnetics 2001;Suppl5:S105-119.
4. Burkhardt M, Spinelli Y, Kuster N: Exposure setup to test effects of wirelesscommunications systems on the CNS. Health Phys 1997;73:770-778.
5. Imaida K, Taki M, Yamaguchi T, et al.: Lack of promoting effects of the
electromagnetic near-field used for cellular phones (929.2 MHz) on rat liver
carcinogenesis in a medium-term liver bioassay. Carcinogenesis
1998;19:311-314.
6. Imaida K, Taki M, Watanabe S, et al.: The 1.5 GHz electromagnetic
near-field used for cellular phones does not promote rat liver carcinogenesis
in a medium-term liver bioassay. Jpn J Cancer Res 1998;89:995-1002.
7. Tsurita G, Nagawa H, Ueno S, et al.: Biological and morphological effects onthe brain after exposure of rats to a 1439 MHz TDMA field.
Bioelectromagnetics 2000;21:364-371.
8. Yamaguchi H, Tsurita G, Ueno S, et al.: 1439 MHz pulsed TDMA fields
affect performance of rats in a T-maze task only when body temperature is
elevated. Bioelectromagnetics 2003;24:223-230.
9. Hata K, Yamaguchi H, Tsurita G, et al.: Short term exposure to 1439 MHz
pulsed TDMA field does not alter melatonin synthesis in rats.
Bioelectromagnetics 2005;26:49-53.
10. Hata K, Yamaguchi H, Tsurita G, et al.: Short term exposure to 1439 MHzpulsed TDMA field does not alter melatonin synthesis in rats.
Bioelectromagnetics 2004;26:49-53.
11. Hata K, Ueno S, Yamaguchi H, et al.: Long-term effects on melatonin
synthesis in male rats after exposure to a 1439 MHz TDMA electromagnetic
field. In "Bioelectromagnetics Society Annual Meeting." 2004.
12. Nandi S, Guzman RC, Yang J: Hormones and mammary carcinogenesis inmice, rats, and humans: a unifying hypothesis. Proc Natl Acad Sci U S A
1995;92:3650-3657.
13. Russo IH, Russo J: Mammary gland neoplasia in long-term rodent studiesEnviron Health Perspect 1996;104:938-967.
14. Thun-Battersby S, Mevissen M, Loscher W: Exposure of Sprague-Dawleyrats to a 50-Hertz, 100-microTesla magnetic field for 27 weeks facilitates
mammary tumorigenesis in the 7,12-dimethylbenz[a]-anthracene model of
breast cancer. Cancer Res 1999;59:3627-3633.
15. Fedrowitz M, Westermann J, Loscher W: Magnetic field exposure increasescell proliferation but does not affect melatonin levels in the mammary glandof female Sprague Dawley rats. Cancer Res 2002;62:1356-1363.
16. Stevens RG, Davis S: The melatonin hypothesis: electric power and breascancer. Environ Health Perspect 1996;104 Suppl 1:135-140.
17. Brainard GC, Kavet R, Kheifets LI: The relationship between
electromagnetic field and light exposures to melatonin and breast cancer
risk: a review of the relevant literature. J Pineal Res 1999;26:65-100.
18. Bartsch H, Bartsch C, Seebald E, et al.: Chronic exposure to a GSM-like
signal (mobile phone) does not stimulate the development of DMBA-inducedmammary tumors in rats: results of three consecutive studies. Radiat Res
2002;157:183-190.