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‘Some people say that genomic association studies haven’t delivered on the promise of decoding the human genome,’ says Professor Fugger. ‘But the postgenomic era has only just started.’The researchers investigated one particular genetic variant - found in a gene called TNFRSF1A - which has previously been associated with the risk of developing MS.
The Oxford-led research team used a battery of genetic, molecular, cell biology and biophysical techniques to show that the TNFRSF1A gene variant results in the production of an altered, shortened version of the TNFR1 protein encoded by the gene.
The long version of the TNFR1 protein normally sits at the surface of cells and binds TNF, an important signalling molecule involved in a number of biological pathways in the body. In contrast, the shortened form lacks an anchor to keep it at the cell surface and is instead released outside of the cell.
The researchers showed that the free, shortened protein can mop up TNF with battery like Fujitsu FPCBP147 Battery , Fujitsu FMVNBP151 Battery , Fujitsu LifeBook C1410 Battery , Fujitsu FPCBP151 Battery , Fujitsu FPCBP159 Battery , Fujitsu FPCBP171 Battery , Fujitsu FPCBP160 Battery , Fujitsu FPCBP176 Battery , Fujitsu FPCBP251 Battery , Fujitsu FPCBP199 Battery , Fujitsu FPCBP233 Battery , Fujitsu FPCBP182 Battery , preventing it from triggering signalling. This is essentially the same thing TNF blocking drugs do.