Non-receptor-tyrosine Kinases Integrate Fast Glucocorticoid Signaling in Hippocampal Neurons. Silei Yang‡1 ... Conclusion: Corticosterone-induced changes in neuronal excitability are initiated at the plasma membrane.
neuron receptor sites
In contrast, synaptic coupling of excitatory and inhibitory motor neurons is achieved through a second population of iAChRs specifically localized at postsynaptic sites on inhibitory motor neurons. Loss of ACR-12 iAChRs from& ...
“If we could find a drug that attaches itself to this site and locks together NMDA (N-methyl-D-aspartate) receptor subunits, that would be huge for fighting disability from stroke and Alzheimer`s and other neurodegenerative diseases.” Published in ... By locking the subunits together, researchers were able to markedly reduce NMDA activity and, subsequently, substantially reduce the amount of calcium that enters neurons in response to the neurotransmitter glutamate.
Involvement of the GluN2A and GluN2B Subunits in Synaptic and Extrasynaptic N-methyl-d-aspartate Receptor Function and Neuronal Excitotoxicity*. Xianju Zhou‡,§,¶1,; Qi Ding‡, ... NMDA receptors (NMDAR). Previous studies have suggested that GluN2A and GluN2B may differentially mediate NMDAR function at synaptic and extrasynaptic locations and play opposing roles in excitotoxicity, such as neurodegeneration triggered by ischemic stroke and brain injury.
Non-receptor-tyrosine Kinases Integrate Fast Glucocorticoid Signaling in Hippocampal Neurons. Silei Yang‡1 ... Conclusion: Corticosterone-induced changes in neuronal excitability are initiated at the plasma membrane.
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